Androgenetic alopecia (AGA) in women, also commonly known as Female pattern hair loss (FPHL) is a nonscarring progressive thinning of hair, particularly in the frontal, central, and parietal scalp, as a result of follicular miniaturisation. Numerous studies have demonstrated the unavoidable connection between androgenetic alopecia in males and coronary artery disease. However, a paucity of similar studies in women calls for the need to know a similar association.

Corelating, different grades of FPHL with the severity of atherosclerosis in healthy, young, non-obese women, would help in isolating women who are prone to developing coronary artery disease (CAD). There is abundance of evidence to prove the relationship between concentrations of lipids and lipoproteins with the development of CAD leading to ischaemic heart disease. It has been demonstrated that estimating the atherogenic index, or the log of the ratio of triglycerides to high-density lipoproteins (HDL), is a superior predictor of cardiovascular events.

Numerous circulating markers of inflammation have been studied for their potential to forecast the risk of vascular events because there is a wealth of evidence linking inflammation to atherosclerosis and acute coronary syndromes (ACS). The biomarker that is most researched in this context is C-reactive protein. It is a prognostic indicator for cardiovascular mortality and recurrence for both primary and secondary protection.

Additionally, estimation of Epicardial Adipose Tissue (EAT) Thickness provides a major indicator of atherosclerosis linked to cardiovascular risk factors and outcomes.

This study would help in identifying young women who otherwise do not have any disease and are still prone to developing CAD. Diagnosing FPHL and correlating it with atherogenic potential would be a non-invasive way to diagnose CAD earlier.