Pterygium is a wing shape fibrovascular degeneration of subconjunctival tissue. It destroys bowman’s membrane and corneal stroma by proliferating as vascularized granulation tissue. Ultraviolet B the main etiological factor mutates p53 tumor suppressor gene present at limbal stem cell and leads to overexpression of TGF-beta and VEGF which results in upregulation of collagenase, cellular migration and angiogenesis. Elastotic degeneration of collagen and subepithelial fibrovascular tissue are the characteristic pathological feature of the pterygium. Pterygia are thick and vascular when early but becomes pale and thin when cease to progress       and leads to formation of dense fibrous tissues and development of considerable corneal astigmatism. Pterygium exerts a tractional force on cornea, which results in flattening of corneal curvature   in horizontal meridian leading to with-the-rule astigmatism. Flattening also occurs due to pooling of tears near the head of pterygium. Most patient of pterygium complaints of foreign body sensation, lacrimation, redness, dryness and itching. Spatial contrast sensitivity and glare disability are worsened in patients even when the visual acuity is minimally affected. Cosmetic reasons, visual impairment, motility     restriction is some of the indication for pterygium surgery.

Pterygium excision is the mainstay of treatment but with high tendency to                          reoccur. The gold standard in treatment of pterygium is pterygium excision with autograft implantation which has low rate of recurrences and complication. The present study aims to evaluate keratometry readings and corneal astigmatic changes following pterygium excision with conjunctival autograft transplantation using fibrin glue. Since a few studies have been done on this aspect therefore this study has been undertaken.