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CTRI Number  CTRI/2025/02/080893 [Registered on: 19/02/2025] Trial Registered Prospectively
Last Modified On: 17/02/2025
Post Graduate Thesis  Yes 
Type of Trial  Interventional 
Type of Study   Surgical/Anesthesia
Diagnostic
Preventive 
Study Design  Randomized, Parallel Group Trial 
Public Title of Study   Donor-Derived Cell-Free DNA: A Breakthrough Biomarker for Early Rejection Detection and Immunosuppression Management in Liver Transplant Patients 
Scientific Title of Study   DONOR-DERIVED CIRCULATING CELL-FREE DNA AS A REJECTION BIOMARKER AND IMMUNOSUPPRESSION MODULATION TOOL IN LIVER TRANSPLANTATION 
Trial Acronym  NIL 
Secondary IDs if Any  
Secondary ID  Identifier 
NIL  NIL 
 
Details of Principal Investigator or overall Trial Coordinator (multi-center study)  
Name  Dr Dinesh Balakrishnan 
Designation  Clinical Professor- Department of Gastrointestinal surgery 
Affiliation  Amrita Institute of Medical Sciences and Research Centre: Amrita Institute of Medical Sciences 
Address  Department of Gastrointestinal Surgery and Solid Organ Transplantation, G-block:2nd floor, Amrita institute of Medical Sciences and Research, Ponekkara P.O, Ernakulam, Pin-682041

Ernakulam
KERALA
682041
India 
Phone  9946861474  
Fax    
Email  dineshb@aims.amrita.edu  
 
Details of Contact Person
Scientific Query
 
Name  Dr.Sudhindran S 
Designation  Chief Transplant Surgeon-Department of Gastroinetstinal Surgery 
Affiliation  Amrita Institute of Medical Sciences and Research Centre: Amrita Institute of Medical Sciences 
Address  G-block,2nd floor, Room No:10 Amrita Institute of Medical Sciences and Research, Ponekkara P.O, Ernakulam, Pin-682041

Ernakulam
KERALA
682041
India 
Phone  7306716242  
Fax    
Email  sudhisalini@icloud.com  
 
Details of Contact Person
Public Query
 
Name  Dr. Lakshmi.V.U 
Designation  Research scholar- Department of Gastrointestinal surgery 
Affiliation  Amrita Institute of Medical Sciences and Research Centre: Amrita Institute of Medical Sciences 
Address  Department of Gastrointestinal surgery-block, 2nd floor Amrita Institute of Medical Sciences and Research, Ponekkara P.O, Ernakulam, Pin-682041

Ernakulam
KERALA
682041
India 
Phone  8304023632  
Fax    
Email  laxmisadhi123@gmail.com  
 
Source of Monetary or Material Support  
Amrita Centre for NanoSciences and Molecular Medicine, B-block, 1st floor, Amrita Institute of Medical Sciences and Research, Ponekkara P.O, Ernakulam, Pin-682041 
The Liver Transplant Society-India, Room No:1246,2nd floor Indraprastha,Apollo Hospotal,Sarita Vihar, New Delhi, Pin-110076 
 
Primary Sponsor  
Name  Liver Transplant Society-India 
Address  Room number 1246, 2nd Floor Indraprastha Apollo Hospital Sarita Vihar New Delhi 110076 India 
Type of Sponsor  Research institution and hospital 
 
Details of Secondary Sponsor  
Name  Address 
Amrita institute of medical sciences and research  Amrita institute of medical sciences, Ponekkara P.O, Ernakulam, Pin-682041 
 
Countries of Recruitment     India  
Sites of Study  
No of Sites = 1  
Name of Principal Investigator  Name of Site  Site Address  Phone/Fax/Email 
Dr Dinesh Balakrishnan  Amrita Institute of Medical Sciences Ethics Committee  Department of Gastrointestinal Surgery and Solid Organ Transplantation, G-block,2nd floor, Room No:10 Amrita institute of medical sciences, Ponekkara P.O, Ernakulam, Pin-682041
Ernakulam
KERALA 
8304023632

dineshb@aims.amrita.edu 
 
Details of Ethics Committee  
No of Ethics Committees= 1  
Name of Committee  Approval Status 
Amrita Institute of Medical Sciences Ethics Committee  Approved 
 
Regulatory Clearance Status from DCGI  
Status 
Not Applicable 
 
Health Condition / Problems Studied  
Health Type  Condition 
Patients  (1) ICD-10 Condition: O||Medical and Surgical,  
 
Intervention / Comparator Agent  
Type  Name  Details 
Intervention  donor-derived cell-free DNA and liver function tests  Blood samples (9 ml) were collected on postoperative days 3, 7, 14, 30, and 90, and dd-cfDNA was quantified. If dd-cfDNA levels were found to be elevated, suggesting a possible early rejection, immunosuppression was adjusted as follows: 1.The primary goal of immunosuppression modulation was to maintain Tacrolimus (TAC) blood levels at 8 ng/ml. If TAC levels were already at this target, immunosuppressive therapy was intensified by either increasing the dosage or adding alternative immunosuppressants such as steroids or Mycophenolate Mofetil (MMF). 2.The steroid dose was increased by additional 10 mg of prednisolone, or Mycophenolate Mofetil was increased to 500 mg thrice daily. Adjustments were made based on platelet and total WBC counts. 3. If dd-cfDNA levels were elevated after postoperative day 30 and the recipient was already on adequate triple immunosuppression, an mTOR inhibitor (Everolimus) was added. This was avoided within the initial 30 days post-transplant due to the risk of impaired wound healing. 4.Granzyme-B levels were to be done in patients who underwent a rejection to differentiate antibody-mediated rejection, so that each type of rejection could be treated accordingly. Simultaneously, if liver function tests (LFTs) showed significant abnormalities, particularly a more than two-fold rise in LFTs without evident causes such as sepsis or graft-related issues (e.g., biliovascular problems), a liver biopsy was performed to confirm rejection. The biopsy results were used to calculate the Rejection Activity Index (RAI) to quantify rejection severity. Biopsy-proven rejection was treated according to the units protocol with Methylprednisolone pulse doses of 10 mg/kg for 3-5 days. If dd-cfDNA levels were normal, patients continued with standard immunosuppressive therapy and were followed up regularly to ensure stable transplant function. dd-cfDNA analysis included assessment of parameters like total cfDNA, dd-cfDNA percentage, integrity ratio, T-cell markers, and Granzyme-B to differentiate between infection, T-cell-mediated rejection, antibody-mediated rejection, and normal profiles. This facilitated precise identification of the presence and type of rejection in each patient. This intervention is to be carried out in each subject over a period of 3 months and then the morphology of allografts in both groups will be compared with CT/MRI or Fibro Scan. The total duration of the study will be 2 years. 
Comparator Agent  Liver function tests alone  Patients were monitored using standard liver function tests alone, according to standard protocol. This included liver function tests daily for the initial 2 weeks post-transplantation. Then these tests were done twice a week, then once a week, then once in 2 weeks and later on monthly basis for a year, unless any complication was suspected, and additional testing is required in that case. Biopsies were performed if there was a two-fold rise in LFTs without evident causes such as sepsis or graft-related issues (e.g., biliovascular problems). This intervention is to be carried out in each subject over a period of 3 months and then the morphology of allografts in both groups will be compared with CT/MRI or Fibro Scan. The total duration of the study will be 2 years. 
 
Inclusion Criteria  
Age From  18.00 Year(s)
Age To  80.00 Year(s)
Gender  Both 
Details  All adults admitted to undergo liver transplantation 
 
ExclusionCriteria 
Details  Multi-organ transplantation, identical twin donor and recipient, ABO-incompatible transplantation 
 
Method of Generating Random Sequence   Computer generated randomization 
Method of Concealment   Sequentially numbered, sealed, opaque envelopes 
Blinding/Masking   Not Applicable 
Primary Outcome  
Outcome  TimePoints 
Primary Outcome: Efficacy of dd-cfDNA in indicating adequate immunosuppression levels
 
Post-operative day 3, 7, 14,30 and 90 
 
Secondary Outcome  
Outcome  TimePoints 
a) Early detection of allograft rejection compared to liver function tests.  Post-operative day3,7,14,30 and 90 
b) Correlation of Granzyme-B levels and antibody mediated rejection  Post-operative day 3,7,14,30 and 90 
c)Comparison of graft morphology 1-year post-transplant in both groups  1-year post-transplantation 
 
Target Sample Size   Total Sample Size="136"
Sample Size from India="136" 
Final Enrollment numbers achieved (Total)= "Applicable only for Completed/Terminated trials"
Final Enrollment numbers achieved (India)="Applicable only for Completed/Terminated trials" 
Phase of Trial   Phase 4 
Date of First Enrollment (India)   28/02/2025 
Date of Study Completion (India) Applicable only for Completed/Terminated trials 
Date of First Enrollment (Global)  Date Missing 
Date of Study Completion (Global) Applicable only for Completed/Terminated trials 
Estimated Duration of Trial   Years="2"
Months="0"
Days="0" 
Recruitment Status of Trial (Global)   Open to Recruitment 
Recruitment Status of Trial (India)  Open to Recruitment 
Publication Details   N/A 
Individual Participant Data (IPD) Sharing Statement

Will individual participant data (IPD) be shared publicly (including data dictionaries)?  

Response - NO
Brief Summary  

Following liver transplantation, acute cellular rejection remains a significant challenge, affecting 10-40% of recipients despite advancements in immunosuppressive therapies. While liver biopsy is the gold standard for diagnosing rejection, it is invasive, costly, and associated with potential complications. Donor-derived cell-free DNA (dd-cfDNA) has emerged as a promising noninvasive biomarker, providing real-time insights into graft health through simple blood sampling. This randomized controlled trial aims to evaluate the efficacy of dd-cfDNA-modulated immunosuppression in reducing acute T-cell mediated rejection. Participants undergoing live donor liver transplantation will be randomized into two groups. Immediately after liver transplantation, both groups of patients will initially be started on immunosuppression as follows :

  • Tacrolimus will be started at 2mg/day in a divided or single dose depending on the formulation to aim for a trough level of 8ng/ml. The dosage will be altered depending on the renal function.
  • Methylprednisolone will be started at 250 mg IV on Day 1, 125mg IV on Day 2,  80mg IV on Day 3, and 40 mg IV on Day 4 . Subsequently, oral prednisolone is started at 30mg daily and tapered weekly by 5mg and stopped at 6 weeks.
  • Mycophenolate Mofetil (MMF) will be started at 500 mg twice daily from day 1, provided the platelet count is more than 25,000/mm3. It will be stopped at 4-6 months. Due to the complexity of the interaction between immunosuppressants and other drugs and hepatic and renal function, some degree of immunosuppression tweaking will be left to the physician’s discretion.

On postoperative day 3, the participants will be randomly assigned into two groups using a computerized randomization technique. The assigned group will be enclosed in sealed, opaque envelopes opened only by the study coordinator.

Group 1 (Test group): Blood samples (9ml) will be collected at the post-operative days 3,7,14,30 & 90 and the dd-cfDNA will be quantified. At any point during the analysis, if dd-cfDNA levels are found to be elevated, this possibly suggests an early rejection, and immunosuppression will be adjusted as follows:

  • Immunosuppression modulation aims to maintain Tacrolimus (TAC) blood levels at 8 ng/ml. If TAC levels are already at this target level, immunosuppressive therapy will be intensified by either increasing the dosage or adding alternative immunosuppressants such as steroids or Mycophenolate mofetil (MMF) depending on clinical parameters
  • Steroid dose will be increased by 10mg of prednisolone or Mycophenolate mofetil will be increased to 500mg thrice a day.
  • This will be based on the platelet count and total WBC count. If the dd-cfDNA levels are elevated on postoperative day 30 or 90, and the recipient is already on adequate triple immunosuppression, an mTOR inhibitor (Everolimus) will be added. This is a relative contraindication within the initial 30 days of transplant due to poor wound healing.

Simultaneously, if liver function tests (LFTs) show significant abnormalities, particularly a more than two-fold rise in LFTs without evident causes like sepsis or graft-related issues (such as biliovascular problems), a liver biopsy will be performed to confirm a rejection. The biopsy results will be used to calculate the Rejection Activity Index (RAI) to quantify the severity of the rejection. Biopsy-proven rejection will be treated according to the unit protocol: methylprednisolone pulsed doses at 10 mg/kg for 3-5 days. If dd-cfDNA levels are found to be normal, the patient will continue with standard immunosuppressive therapy, with regular follow-up to ensure stable transplant function.

dd-cfDNA analysis will involve assessing parameters like total cfDNA, dd-cfDNA percentage, integrity ratio, T-cell markers, and Granzyme-B to differentiate between infection, T-cell-mediated rejection, antibody-mediated rejection, and a normal profile. This will help precisely identify the presence and type of rejection in each patient.

Group 2(Standard group): Patients are monitored using standard liver function tests alone. The biopsy will be performed if there is a two-fold rise in liver function tests (aminotransferases, alkaline phosphatase, gamma-glutamyl transferases) without evident causes like graft-related issues (such as biliovascular problems) or sepsis (based on elevated WBC, CRP or procalcitonin, and culture reports) Biopsy-proven rejection will be treated as elaborated in Group 1.

Steroid-resistant rejection is defined as a form of acute rejection in which the liver transplant recipient does not respond adequately to three aliquots of pulsed steroids This will be confirmed by liver biopsy and treated usually by Antithymocyte globulin (ATG). The measurement of the study outcome will be the number of biopsy-proven rejections in the test group & standard group.

We have completed a pilot study as per our previous protocol with CTRI registration.The study included a total of 50 patients, with 25 allocated to the standard care group and 25 to the test group. In the test group, 8 patients experienced allograft rejection, of whom 4 cases were confirmed via liver biopsy (T-cell-mediated rejection and antibody-mediated rejection). Biopsy could not be performed in the remaining 4 patients due to complications such as sepsis and thrombocytopenia. In the standard group, 6 patients experienced allograft rejection, with 4 cases confirmed by liver biopsy (all T-cell-mediated rejection). Among the 8 patients who experienced rejection in the test group, dd-cfDNA levels increased before abnormalities in liver function tests (LFTs) in 3 patients, with elevations occurring 5 days, 6 days, and 20 days before LFT changes were detected. Exclusions were necessary for 3 patients from the standard group and 1 from the test group due to mortality within 75 days post-transplantation. Importantly, there were no false-positive or false-negative results in rejection detection using dd-cfDNA.Among patients who did not experience rejection, dd-cfDNA levels steadily declined from approximately 10% on postoperative day 7 to less than 1% by day 180. In contrast, a marked elevation in dd-cfDNA levels was observed in patients undergoing rejection. Additionally, Granzyme-B levels were significantly elevated during antibody-mediated rejection, rising from the normal range of 20–40 pg/mL to 140 pg/mL.

 


 
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